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AOD-9604

Anti-Obesity Drug 9604 — C-Terminal GH Fragment

GH-derived fat-targeting fragment — specifically activates lipolysis in adipose tissue without GH's growth-promoting effects.

Tier C — Early or Mixed: Limited human trials; effect size uncertain. How our tiers work →
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Research facts

Half-life~2h
RouteSubQ / Nasal / Oral
Typical dose250–500 mcg/day
FrequencyOnce daily (morning fasted)
SolventBacteriostatic water
pH5.5–7.5
Stability30 days at +4°C

Mechanism of action

AOD-9604 (Tyr-hGH fragment 177-191) is derived from the C-terminal region of human Growth Hormone responsible for its fat-metabolising activity, specifically engineered to activate lipolysis without the growth-promoting or glucose-altering effects of full HGH. It stimulates lipolytic pathways in adipose tissue via a β3-adrenergic receptor-like mechanism, activating hormone-sensitive lipase and suppressing lipogenesis (via inhibition of acetyl CoA carboxylase). Unlike HGH, AOD-9604 does not activate IGF-1 production or insulin-like signalling, avoiding the metabolic side effects of full GH. FDA granted it GRAS (Generally Recognised as Safe) status for use as a food additive — an unusual distinction reflecting favourable safety data. Phase IIb trials showed modest fat loss vs. placebo in obese subjects.

Documented effects (research-model)

  • Targeted adipose lipolysis without IGF-1 activation
  • Fat loss without glucose or growth effects of HGH
  • FDA GRAS designation (food ingredient safety)
  • Cartilage repair research — documented in osteoarthritis models
  • Nasal and oral bioavailability (route flexibility)

Research protocols

ProtocolDoseFrequencyCycleVial
Standard 250–500 mcg SubQ or nasal Once daily morning fasted 12 weeks 2mg / 2mL BAC water

Research context only. Not medical advice. Consult a qualified healthcare professional before any protocol decision.

Synergies

  • Tesamorelin MODERATE
    Both target fat but via different mechanisms: AOD-9604 is direct lipolytic; Tesamorelin is GH-mediated. Avoid redundancy.
  • BPC-157 MODERATE
    AOD-9604 shows cartilage repair potential; BPC-157 drives connective tissue healing. Joint recovery combination.

Myths & misconceptions

Myth AOD-9604 is as effective as semaglutide for fat loss.
Reality Phase IIb data for AOD-9604 showed modest, statistically borderline improvements. GLP-1 agonists like semaglutide show 15%+ body weight reduction in RCTs. AOD-9604 has a weaker evidence base for primary fat loss.

Evidence gaps

  • Phase III fat loss trial was never completed after mixed Phase IIb results.
  • Cartilage repair claims are primarily from in vitro and animal studies — not established in human trials.
  • Long-term lipolytic effect with continuous use (tolerance development) not studied.

Safety notes

FDA GRAS status for food use. Well-tolerated in human trials with no significant adverse events. Monitor fasting glucose (though less concern than full HGH). Research context only.

Biomarkers to monitor

Fasting GlucoseHbA1cTriglyceridesTotal Cholesterol

Primary-source citations

  • PMID 16698119 — AOD9604: a modified fragment of human growth hormone with lipolytic and antiobesity properties (Endocrinology. 2006)

Frequently asked

What is AOD-9604?

AOD-9604 (Anti-Obesity Drug 9604 — C-Terminal GH Fragment) is a research compound classified in our library as Tier C — Limited human trials; effect size uncertain.. GH-derived fat-targeting fragment — specifically activates lipolysis in adipose tissue without GH's growth-promoting effects.

What is the evidence tier for AOD-9604?

We classify AOD-9604 as Tier C: Limited human trials; effect size uncertain. See our full peptide evidence tiers explainer for how we assign S/A/B/C/D.

What is the research dose of AOD-9604?

For AOD-9604, typical research dose is 250–500 mcg/day, route is SubQ / Nasal / Oral, half-life is ~2h. Protocols vary by research goal — see the protocols section on this page for standard and advanced dosing schedules. Research use only, not medical advice.

Is AOD-9604 safe?

FDA GRAS status for food use. Well-tolerated in human trials with no significant adverse events. Research context only — no compound on this site is approved for human therapeutic use unless explicitly noted.

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