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Tier D Cognitive

Dihexa

N-hexanoic-Tyr-Ile-(6) aminohexanoic amide — HGF Potentiator

The most potent synaptogenic compound discovered — 7 orders of magnitude more potent than BDNF in animal models.

Tier D — Preclinical Only: Animal or in-vitro data only. How our tiers work →

Research facts

Half-life~24h (estimated)
RouteOral / Transdermal / SubQ
Typical dose1–10 mg/day (no established human dose)
FrequencyOnce daily
SolventVarious
pHN/A
StabilityStable at room temperature

Mechanism of action

Dihexa is an orally bioavailable, brain-penetrant small peptide derived from angiotensin IV. It acts as a potent hepatocyte growth factor (HGF) potentiator — stabilising HGF and dramatically enhancing its binding to the Met receptor (c-Met). This drives synaptogenesis (new synapse formation) in the hippocampus and other brain regions. In rats, Dihexa was found to be ~7 orders of magnitude more potent than BDNF in promoting spine density and dendrite branching. It dramatically improved cognitive function in aged rodents. However, c-Met is also a proto-oncogene — Met amplification drives multiple cancers, raising significant theoretical cancer safety concerns.

Documented effects (research-model)

  • Most potent known synaptogenic compound in animal models
  • Cognitive improvement in aged rodent models
  • Oral/transdermal bioavailability (unusual for a peptide)
  • HGF/Met-mediated neuroplasticity amplification

Research protocols

ProtocolDoseFrequencyCycleVial
Highly Experimental 1–10 mg/day (no consensus) Once daily Short cycles (4 weeks max based on c-Met concerns) Various

Research context only. Not medical advice. Consult a qualified healthcare professional before any protocol decision.

Synergies

No documented synergies in the current reference set.

Myths & misconceptions

Myth Dihexa is definitely safe because it's "just a peptide".
Reality Dihexa activates the c-Met receptor — a known oncogene. HGF/Met signalling drives proliferation in multiple cancer types. The safety profile in humans over extended periods is completely unknown. The extraordinary potency that makes it exciting also makes safety evaluation critical.

Evidence gaps

  • No human clinical trials.
  • c-Met is a proto-oncogene — cancer safety not characterised.
  • Optimal dose, dosing schedule, and cycle length completely unknown in humans.

Safety notes

EXTREME CAUTION: c-Met activation has carcinogenic potential. Absolutely contraindicated in personal or family history of cancer. No established safe dose in humans. This is among the most experimental and potentially high-risk compounds in this library despite its extraordinary preclinical profile.

Biomarkers to monitor

Full Blood CountLiver function testsPSA (prostate specific antigen)

Primary-source citations

  • PMID 23143601 — Dihexa: a potent pro-cognitive HGF potentiator derived from angiotensin IV (J Pharmacol Exp Ther. 2013)

Frequently asked

What is Dihexa?

Dihexa (N-hexanoic-Tyr-Ile-(6) aminohexanoic amide — HGF Potentiator) is a research compound classified in our library as Tier D — Animal or in-vitro data only.. The most potent synaptogenic compound discovered — 7 orders of magnitude more potent than BDNF in animal models.

What is the evidence tier for Dihexa?

We classify Dihexa as Tier D: Animal or in-vitro data only. See our full peptide evidence tiers explainer for how we assign S/A/B/C/D.

What is the research dose of Dihexa?

For Dihexa, typical research dose is 1–10 mg/day (no established human dose), route is Oral / Transdermal / SubQ, half-life is ~24h (estimated). Protocols vary by research goal — see the protocols section on this page for standard and advanced dosing schedules. Research use only, not medical advice.

Is Dihexa safe?

EXTREME CAUTION: c-Met activation has carcinogenic potential. Absolutely contraindicated in personal or family history of cancer. Research context only — no compound on this site is approved for human therapeutic use unless explicitly noted.

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