Home / Compounds / Hexarelin
Tier B Growth Hormone

Hexarelin

Examorelin — Most Potent GHRP

The most potent GHRP in terms of GH peak — also has direct cardiac protective effects via CD36 receptor, independent of GH.

Tier B — Promising: Some human data + strong preclinical evidence. How our tiers work →

Research facts

Half-life~2–3h
RouteSubQ / IV
Typical dose100–200 mcg
Frequency2–3× daily
SolventBacteriostatic water
pH5.5–7.5
Stability30 days at +4°C

Mechanism of action

Hexarelin is the most potent GHRP in terms of GH pulse amplitude — achieving higher peak GH than GHRP-2, GHRP-6, or Ipamorelin at equivalent doses. Like other GHRPs, it activates GHS-R1a. However, hexarelin also binds the scavenger receptor CD36 on cardiac myocytes and macrophages, producing cardioprotective effects independent of GH or IGF-1. This direct cardiac action (anti-apoptotic, anti-inflammatory in cardiac tissue) has been demonstrated in multiple animal models of myocardial infarction and heart failure. Human Phase I/II data exist primarily for its GH-stimulatory effects.

Documented effects (research-model)

  • Highest GH pulse amplitude among GHRPs
  • Cardioprotection via CD36 receptor (independent of GH)
  • Anti-apoptotic effects in cardiac myocytes
  • Phase II human GH deficiency data
  • Research tool for cardiac protection models

Research protocols

ProtocolDoseFrequencyCycleVial
Standard 100–200 mcg SubQ 2–3× daily 8–12 weeks 5mg / 5mL BAC water

Research context only. Not medical advice. Consult a qualified healthcare professional before any protocol decision.

Synergies

  • CJC-1295 HIGH
    GHRH + GHRP synergy. Hexarelin's extreme GH peak is further amplified by GHRH co-administration.

Myths & misconceptions

Myth Hexarelin's cardiac effects are clinically proven.
Reality Hexarelin's direct cardiac effects via CD36 are mechanistically compelling and well-established in animal models, but human clinical trials for cardiac protection are in early stages. The evidence is promising but not yet practice-changing.

Evidence gaps

  • Desensitisation occurs faster with hexarelin than other GHRPs — unclear optimal cycle length.
  • Human cardiac protection trials not yet completed.
  • Cortisol and prolactin co-stimulation at high doses.

Safety notes

Rapid receptor desensitisation — efficacy decreases with chronic daily use more than other GHRPs. Cortisol and prolactin elevation. Monitor all GH-axis markers plus cardiac function in research protocols.

Biomarkers to monitor

IGF-1GHCortisolProlactinCreatine Kinase (cardiac marker)

Primary-source citations

  • PMID 15456933 — Hexarelin protects against ischaemic myocardial damage via CD36 receptor (Eur J Pharmacol. 2004)

Frequently asked

What is Hexarelin?

Hexarelin (Examorelin — Most Potent GHRP) is a research compound classified in our library as Tier B — Some human data + strong preclinical evidence.. The most potent GHRP in terms of GH peak — also has direct cardiac protective effects via CD36 receptor, independent of GH.

What is the evidence tier for Hexarelin?

We classify Hexarelin as Tier B: Some human data + strong preclinical evidence. See our full peptide evidence tiers explainer for how we assign S/A/B/C/D.

What is the research dose of Hexarelin?

For Hexarelin, typical research dose is 100–200 mcg, route is SubQ / IV, half-life is ~2–3h. Protocols vary by research goal — see the protocols section on this page for standard and advanced dosing schedules. Research use only, not medical advice.

What does Hexarelin stack well with?

Hexarelin stacks well with CJC-1295. GHRH + GHRP synergy. Hexarelin's extreme GH peak is further amplified by GHRH co-administration.

Is Hexarelin safe?

Rapid receptor desensitisation — efficacy decreases with chronic daily use more than other GHRPs. Cortisol and prolactin elevation. Research context only — no compound on this site is approved for human therapeutic use unless explicitly noted.

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