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LL-37

Human Cathelicidin Antimicrobial Peptide — hCAP18 Active Fragment

The body's native antimicrobial peptide — kills bacteria, viruses, fungi, and biofilms while modulating immune signalling.

Tier D — Preclinical Only: Animal or in-vitro data only. How our tiers work →
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Research facts

Half-life~2–4h
RouteSubQ / Topical / Nasal
Typical dose25–100 mcg/day
FrequencyOnce or twice daily
SolventBacteriostatic water
pH5.5–7.5
Stability30 days at +4°C

Mechanism of action

LL-37 is the C-terminal active fragment of the human cathelicidin hCAP18 — the only member of the cathelicidin family in humans. It is an amphipathic alpha-helical peptide that disrupts microbial membranes via electrostatic interaction and insertion into lipid bilayers, causing membrane permeabilisation and death. Its antimicrobial spectrum includes gram-positive bacteria (MRSA, Streptococcus), gram-negative bacteria (E. coli, Pseudomonas), fungi, and enveloped viruses. Beyond direct killing, LL-37 activates TLR signalling, recruits neutrophils and macrophages, induces dendritic cell maturation, and has documented anti-biofilm activity against chronic biofilm-forming pathogens (a major limitation of conventional antibiotics). It also promotes wound healing, angiogenesis, and re-epithelialisation via EGF receptor transactivation.

Documented effects (research-model)

  • Broad-spectrum antimicrobial (bacteria, fungi, viruses)
  • Anti-biofilm activity against antibiotic-resistant pathogens
  • Immune cell recruitment and activation
  • Wound healing via EGF receptor transactivation
  • Anti-inflammatory dual role (context-dependent)
  • Topical and systemic infection defence research

Research protocols

ProtocolDoseFrequencyCycleVial
Standard 50 mcg/day SubQ Once daily 4–8 weeks 5mg / 5mL BAC water

Research context only. Not medical advice. Consult a qualified healthcare professional before any protocol decision.

Synergies

  • Thymosin Alpha-1 MODERATE
    LL-37 provides innate antimicrobial killing; TA-1 enhances adaptive immune response. Complementary immune defence.

Myths & misconceptions

Myth LL-37 can replace antibiotics for bacterial infections.
Reality LL-37 has demonstrated antimicrobial activity in vitro, but translating this to clinical infection treatment is not established in human trials. Antibiotic resistance mechanisms exist for LL-37, and therapeutic concentrations at infection sites are hard to achieve.

Evidence gaps

  • No completed human RCTs for LL-37 as an antimicrobial therapeutic.
  • Optimal delivery route for different infection types is not established.
  • Pro-inflammatory effects at high concentrations (LL-37 can worsen inflammatory conditions like psoriasis) vs. anti-inflammatory at physiological levels — dose-response not characterised in humans.

Safety notes

Generally well-tolerated topically. Injectable use has limited human safety data. High concentrations may exacerbate inflammatory conditions (psoriasis, atopic dermatitis). Research use only.

Biomarkers to monitor

Full Blood CountWBC differentialhs-CRP

Primary-source citations

  • PMID 16091480 — Antimicrobial peptides and innate immunity (Nat Rev Immunol. 2005)

Frequently asked

What is LL-37?

LL-37 (Human Cathelicidin Antimicrobial Peptide — hCAP18 Active Fragment) is a research compound classified in our library as Tier D — Animal or in-vitro data only.. The body's native antimicrobial peptide — kills bacteria, viruses, fungi, and biofilms while modulating immune signalling.

What is the evidence tier for LL-37?

We classify LL-37 as Tier D: Animal or in-vitro data only. See our full peptide evidence tiers explainer for how we assign S/A/B/C/D.

What is the research dose of LL-37?

For LL-37, typical research dose is 25–100 mcg/day, route is SubQ / Topical / Nasal, half-life is ~2–4h. Protocols vary by research goal — see the protocols section on this page for standard and advanced dosing schedules. Research use only, not medical advice.

Is LL-37 safe?

Generally well-tolerated topically. Injectable use has limited human safety data. Research context only — no compound on this site is approved for human therapeutic use unless explicitly noted.

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