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NAD+

Nicotinamide Adenine Dinucleotide — IV / SubQ / Nasal Administration

The cellular energy currency and sirtuin activator — declines with age, supplementation supports mitochondrial function, DNA repair, and longevity pathways.

Tier B — Promising: Some human data + strong preclinical evidence. How our tiers work →
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Research facts

Half-lifeMinutes (IV) / 1–4h SubQ equivalent
RouteIV / SubQ / Nasal / Oral (NMN/NR)
Typical dose100–500 mg (IV/SubQ)
FrequencyWeekly IV or daily SubQ nasal
SolventSterile saline (IV) / BAC water (SubQ)
pH7.0–7.4
StabilityUse IV within 24h; SubQ 7 days at +4°C

Mechanism of action

NAD+ (Nicotinamide Adenine Dinucleotide) is a coenzyme present in every living cell, functioning as the central electron carrier in oxidative phosphorylation (accepting H⁺ as NADH in the citric acid cycle and ETC) and as a substrate for sirtuin deacylases (SIRT1–7) and PARP enzymes involved in DNA repair.

NAD+ levels decline 50% between age 40–60. This reduction impairs mitochondrial function (less NADH for ATP synthesis), reduces sirtuin activity (less DNA repair, less metabolic regulation), and compromises PARP-mediated genome maintenance. Restoring NAD+ via direct supplementation or precursors (NMN, NR) aims to reverse these age-associated deficits.

Multiple human RCTs with NMN and NR (oral precursors) demonstrate that NAD+ levels in blood can be meaningfully elevated. IV NAD+ bypasses conversion steps for rapid elevation. David Sinclair (Harvard) and Bryan Johnson both incorporate NAD+ supplementation in documented longevity protocols, though direct injectable NAD+ data is less robust than oral precursor data.

Documented effects (research-model)

  • Mitochondrial function support via NADH provision for ETC
  • Sirtuin (SIRT1, SIRT3) activation for DNA repair and metabolic regulation
  • PARP enzyme substrate for genome integrity maintenance
  • Circadian rhythm regulation via NAMPT pathway
  • Muscle energy metabolism and exercise recovery
  • Cognitive function and neuroprotection in animal models

Research protocols

ProtocolDoseFrequencyCycleVial
SubQ/Nasal 100 mg SubQ daily or nasal spray Daily Ongoing with quarterly breaks 500mg / 5mL sterile water
IV Protocol 250–500 mg IV Weekly or monthly Ongoing wellness protocol IV preparation

Research context only. Not medical advice. Consult a qualified healthcare professional before any protocol decision.

Synergies

  • SS-31 HIGH
    SS-31 optimises ETC function; NAD+ provides the substrate. Direct mitochondrial metabolic synergy.
  • GHK-Cu MODERATE
    Dual anti-ageing pathway: GHK-Cu at tissue gene expression level + NAD+ at cellular energy/sirtuin level.
  • MOTS-c HIGH
    MOTS-c encoded by mitochondrial genome; NAD+ supports mitochondrial function. Synergistic mitochondrial support.

Myths & misconceptions

Myth Oral NMN/NR is just as effective as IV NAD+.
Reality Oral NMN and NR elevate blood NAD+ levels by 40–90% in RCTs. IV NAD+ bypasses gut conversion for faster elevation. Whether the route matters for functional outcomes is not established — oral precursors are far better studied and much less expensive.
Myth NAD+ supplementation will add decades to your life.
Reality NAD+ is a compelling longevity pathway but translating animal lifespan data to humans remains unproven. Current human evidence supports modest functional benefits (energy, muscle recovery) but longevity claims are extrapolations.

Evidence gaps

  • Direct injectable NAD+ (SubQ) lacks robust clinical trial data — most NAD+ trials use oral NMN/NR.
  • Whether blood NAD+ elevation translates to meaningful tissue-level NAD+ increases in humans is debated.
  • Long-term cancer risk of sustained NAD+ elevation is a theoretical concern needing longitudinal study.

Safety notes

IV NAD+ can cause flushing, chest tightness, nausea — must be administered slowly. SubQ generally well-tolerated. Oral precursors (NMN, NR) have superior safety data. Monitor LFTs, kidney function.

Biomarkers to monitor

Full Blood CountALTGGTTriglyceridesFasting GlucoseHbA1c

Primary-source citations

  • PMID 34728542 — Nicotinamide mononucleotide increases muscle insulin sensitivity in prediabetic women (Science. 2021)
  • PMID 33186449 — Long-term NMN administration increases NAD+ levels and improves metabolic function in mice (Nat Metab. 2020)

Frequently asked

What is NAD+?

NAD+ (Nicotinamide Adenine Dinucleotide — IV / SubQ / Nasal Administration) is a research compound classified in our library as Tier B — Some human data + strong preclinical evidence.. The cellular energy currency and sirtuin activator — declines with age, supplementation supports mitochondrial function, DNA repair, and longevity pathways.

What is the evidence tier for NAD+?

We classify NAD+ as Tier B: Some human data + strong preclinical evidence. See our full peptide evidence tiers explainer for how we assign S/A/B/C/D.

What is the research dose of NAD+?

For NAD+, typical research dose is 100–500 mg (IV/SubQ), route is IV / SubQ / Nasal / Oral (NMN/NR), half-life is Minutes (IV) / 1–4h SubQ equivalent. Protocols vary by research goal — see the protocols section on this page for standard and advanced dosing schedules. Research use only, not medical advice.

What does NAD+ stack well with?

NAD+ stacks well with SS-31. SS-31 optimises ETC function; NAD+ provides the substrate. Direct mitochondrial metabolic synergy.

Is NAD+ safe?

IV NAD+ can cause flushing, chest tightness, nausea — must be administered slowly. SubQ generally well-tolerated. Research context only — no compound on this site is approved for human therapeutic use unless explicitly noted.

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